Advanced peptide delivery technology

Most peptides never survive the trip.

Unformulated peptides lose almost everything to gastric acid, mucus and tight junctions — oral bioavailability below 1–2%. The Stasis ION™ System is a protic ionic liquid excipient platform, built from citric acid and amino acids, that carries peptide cargo across mucosal barriers without disrupting them.

>57×Epithelial permeation vs. saline, ex vivo porcine model
<10 nmConfirmed sub-10 nm secondary population (DLS)
5 organsSystemic distribution, 1 h post oral dose (mouse)
4 months101.1–101.9% recovery at room temperature
Non-cytotoxicISO 10993-5, CCK-8 at 24 h & 48 h

The delivery challenge

Six barriers stand between a peptide and the bloodstream

Each one is a separate failure mode. A delivery system that solves only one of them doesn't move the number.

Enzymatic degradation

Proteases throughout the GI tract, nasal mucosa and sublingual epithelium cleave peptide bonds. Gastric acid at pH 1.5–3.5 causes direct hydrolysis before absorption can occur.

The mucus barrier

A viscoelastic hydrogel of cross-linked mucin covers every mucosal surface. Calcium cross-links raise viscosity and trap large molecules before they reach epithelial cells.

Epithelial tight junctions

Claudins and occludins seal the space between cells. Molecules above ~1 kDa cannot pass paracellularly — which excludes most therapeutic peptides at 1–50 kDa.

First-pass metabolism

Peptides absorbed from the gut enter portal circulation and pass through the liver before reaching systemic circulation. Sublingual and nasal routes bypass this entirely.

Peptide aggregation

In aqueous solution peptides form macro-aggregates. Only the outermost surface interacts with tissue — interior molecules stay inaccessible, cutting effective concentration.

Mucociliary clearance

In the nasal cavity, ciliated cells sweep mucus toward the nasopharynx continuously — limiting the absorption window to minutes for non-mucoadhesive formulations.

Platform architecture

A protic ionic liquid built from two things the body already makes

The system forms through a proton transfer reaction between citric acid — a Krebs cycle intermediate — and the amino acids lysine and arginine. The result is a room-temperature liquid with a dense three-dimensional network of electrostatic and hydrogen-bonding interactions around the peptide cargo.

No novel synthetic chemical entities. Every component carries established GRAS and USP/NF status and is metabolised through normal biochemical pathways.

Diagram: citric acid and lysine undergo proton transfer to form a citrate anion and protonated lysine cation, held in a hydrogen-bonded protic ionic liquid network.

Six mechanisms, working together

Permeation enhancement occurs through ionic microenvironment effects and reversible protein–ion interactions — not membrane disruption, lipid extraction or cytotoxic mechanisms.

Mucus fluidization

The citrate anion chelates the Ca²⁺ ions that cross-link mucin fibres, lowering viscosity so the peptide–PIL complex can reach the epithelial surface. Reversible on dilution.

Tight junction modulation

Amino acid cations interact electrostatically with claudin domains, triggering transient actin reorganisation that widens paracellular gaps. TEER returns to baseline afterward.

Mucoadhesion

Cationic groups adhere to negatively charged sialic acid in nasal and sublingual mucosa, extending residence time and counteracting mucociliary clearance.

Protease inhibition

High ionic strength plus local pH modulation reversibly inhibits digestive proteases; citrate sequesters the Ca²⁺ and Zn²⁺ cofactors many proteases depend on.

pH buffering

Citric acid maintains a protective microenvironment through the acidic gastric phase (pH 1.5–3.5), preventing acid-induced hydrolysis of peptide bonds in transit.

Nanoparticle self-assembly

Bimodal distribution: 200–300 nm peptide-loaded vehicles for sustained release, plus a sub-10 nm population small enough to pass through mucus gel pores directly.

Preclinical evidence

What the data shows — and where each number comes from

Findings below are from independent third-party laboratory studies. Class-level figures from the peer-reviewed ionic liquid literature are marked separately and are not measurements of this platform.

Stasis ION™ data
>57×
Epithelial barrier permeation vs. saline control
Ex vivo Franz cell, full-thickness porcine tissue at 0.5 mm — one of the thickest epithelial barriers available. Oral, sublingual and nasal mucosa are roughly 5× thinner, so this figure is a conservative floor, not a ceiling. 4.59% vs 0.08% cumulative permeation at 24 h.
Stasis ION™ data
<10 nm
Sub-10 nm secondary population
5–10 nm confirmed by DLS — below the 100–500 nm pore threshold of mucus gel networks.
Stasis ION™ data
5
Organs reached, one hour post oral dose
FITC-labelled peptide in mice: intestine (localised within villi), liver, lungs, kidneys, stomach. Water control showed negligible signal in every organ.
Stasis ION™ data
101.1–101.9%
HPLC recovery across four months at room temperature
No degradation trend across the full observation period; all four time points inside ICH Q1A(R2) criteria of 90–110%. Plus 105.8% after 3 days at 37 °C and 104.6% after 10 days of intense light. No cold chain required.
Stasis ION™ data
80–100%
Cell viability, CCK-8 assay
Non-cytotoxic per ISO 10993-5 (>70% threshold) at both 24 h and 48 h exposure, with no acute inflammatory indicators.
Class-level reference
Systemic plasma bioavailability increase
From Angsantikul et al., Adv. Funct. Mater. 2020 — a different ionic liquid (choline-glycolate) delivering monoclonal antibodies in rats. Establishes mechanistic precedent for the ionic liquid class. Not a Stasis ION™ measurement.
Class-level reference
Mechanism validated in the peer-reviewed literature
The same CGLY study quantified 45% mucus viscosity reduction (0.577 → 0.318 Pa·s), a 4–5× increase in paracellular transport, and confirmed the paracellular route as the primary mechanism — with TEER returning to baseline after removal. Safe at 625 mg/kg/day over 7 days in rats with normal GI histopathology. These outcomes describe the ionic liquid class, not this formulation.

Delivery routes

One composition, four routes

The same ionic liquid addresses the barriers common to every mucosal and epithelial surface.

01

Oral / gastrointestinalPrimary

Protects the peptide through gastric acid and proteases, fluidizes intestinal mucus and modulates tight junctions. Multi-organ systemic distribution confirmed in the mouse FITC study, with signal localised inside intestinal villi rather than the lumen.

02

Intranasal / mucosalHigh priority

Nasal epithelium is ~0.1 mm against the 0.5 mm test model, and the route bypasses hepatic first-pass metabolism entirely. Mucoadhesive amino acid cations extend residence time against mucociliary clearance.

03

Sublingual / buccalHigh priority

Thin (~0.1 mm), highly vascularised, bypasses first-pass metabolism, and avoids the proteolytic environment of the gut. Mucoadhesion counteracts the tendency of sublingual formulations to be swallowed before absorption.

04

Topical / external skinSupporting

The >57× enhancement was itself measured across full-thickness skin. Suitable where the target is the skin — collagen-stimulating, wound-healing and other bioactive peptides. The platform's primary differentiation remains oral, nasal and sublingual.

Composition & regulatory position

Excipients with an existing regulatory record

Because every component is already accepted across food, pharmaceutical, cosmetic and nutraceutical categories, the pathway is materially shorter than for a novel synthetic delivery system.

Regulatory status of Stasis ION™ System components
ComponentFDA GRASUSP/NFEU food additiveRole in system
Citric acidGRAS (21 CFR 184.1033)MonographE330Anion; Ca²⁺ chelator; pH buffer; mucus fluidizer
L-LysineGRAS (dietary supplement)MonographPermitted amino acidCation; mucoadhesive; tight junction modulator
L-ArginineGRAS (dietary supplement)MonographPermitted amino acidCation; mucoadhesive; tight junction modulator

Application categories

Fully compatible with nutraceutical/dietary supplement (FDA DSHEA, EFSA), cosmetic/cosmeceutical (21 CFR 700, EU Cosmetics Regulation) and research-use-only frameworks. Compatible with pharmaceutical development via the 505(b)(2) pathway using pharmacopoeial excipients.

Manufacturing

Finished product is manufactured at a facility holding NSF/ANSI 455-2 GMP certification for dietary supplements. That certificate covers the facility and its quality systems — it is held by the contract manufacturer, and it is not a certification of the Stasis ION™ System or of any finished product. Certificate details are reproduced in Appendix B of the dossier.

Scope of these findings

We would rather you know the limits up front than discover them in due diligence.

  • No human clinical data exists for this platform. All findings are preclinical. Clinical translation would require IND-enabling studies, Phase I safety evaluation and route-specific pharmacokinetics in human subjects.
  • The in vivo data is qualitative. The oral distribution study confirms multi-organ distribution by fluorescence imaging. It does not provide plasma concentration, absolute bioavailability, or PK parameters.
  • Permeation data is ex vivo, generated in a porcine tissue model; in vivo data was generated in mice. Interspecies differences mean direct extrapolation to human pharmacokinetics requires clinical validation.
  • One model compound. Stability and permeation used a short-chain therapeutic peptide (2–5 amino acids). The stabilisation mechanism is sequence-independent, but formulators working above ~10 kDa should request peptide-specific evaluation.
  • Direct protease inhibition assay data is not included in the preclinical package; that effect is inferred from stability and permeation data and supported by the ionic liquid literature.
  • No claims of therapeutic efficacy, disease treatment or clinical bioavailability are made or implied. The Stasis ION™ System is excipient technology; Stasis does not manufacture drugs.

The full preclinical dossier

52 pages: platform architecture and ionic chemistry, analytical validation and the 1,000× dilution QC method, nanoparticle characterisation, stability studies, the in vivo distribution study, cytotoxicity, the ex vivo permeation model, delivery route applications, study limitations, regulatory positioning, and the complete third-party test report.

Version 1.0 · August 2026 · Confidential — technical document

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